Paulo Eduardo Ocke Reis
1Department of Specialized and General Surgery, Fluminense Federal University, Rio de Janeiro, Brazil
2Vascular Clinic Ocke Reis, Rio de Janeiro, Brazil
Received Date: December 21, 2020; Accepted Date: December 23, 2020; Published Date: December 30, 2020
Citation: Ocke Reis PE (2020) Covered Stents-Historical Short Note. J Vasc Endovasc Ther. 5 No. 6: 32.
The first commercially available covered stent was Corvita®, which was developed in Belgium by Jean Pierre Becquemin [1]. In 1997, we began our experience with corvita, it was a braided elgiloy frame coated by urethane fibers that has been replaced in sequence by Wallgraft. In 2001, I did a monograph “Percutaneous treatment of the vascular trauma” to be holder from the Brazilian college of surgeons (Figure 1).
Covered stents have expanded the use of endovascular procedures [2]; they can be either with Dacron (Wallgraft®) or polytetrafluoroethylene (Solaris®, Viabahn®, iCast® (outside the United States, Advanta® V12), Jostent®, Fluency®) and can be balloon-expandable (iCast® or Advanta® V12) or self-expandable (Solaris®, Viabahn®, Jostent®, Wallgraft®). The treatment of vascular disease has changed dramatically during the last two decades. They are used mainly for the treatment of traumatic arterial lesions [3], arteriovenous fistulas or false aneurysms, peripheral aneurysms and, more recently, for the treatment of obstructive vascular disease of the aortoiliac and femoropopliteal sectors. To treat visceral artery aneurysms (vaas) covered stents can be useful ,when the artery is not so tortuous and the vaas are not intraparenquimal [4].The major technical limitations to implant it in vaas are severe tortuosity or sometimes small caliber arteries. There are a lot of available stent grafts, we are getting experience, now, with the new brazilian covered stent, Solaris® (Figures 2-5) This stent-graft is more radiopaque than other conventional nitinol stents available in Brazil, it is a flexible selfexpanding stent graft with PTFE. Those stents are a barrier to the ingrowth of neointimal hyperplasia, sealing off the inflammatory surface, and thus have the potential to inhibit restenosis [5].