Endovascular Management of Acute Pulmonary Embolus

Reis PEO*, Roever L and Reis IFO


DOI10.21767/2573-4482.100011
Reis PEO1,3*, Roever L2 and Reis IFO3

1Fluminense Federal University, Rio de Janeiro, Brazil

2Federal University of Uberlândia, Brazil

3Vascular Clinic Ocke Reis, Rio de Janeiro, Brazil

*Corresponding Author:
Reis PEO
Department of Specialized and General Surgery Fluminese Federal University, Rio de Janeiro, Brazil
Tel: +55 21 2629 5000
E-mail: vascular@pauloocke.com.br

Received date: May 09, 2016; Accepted date: May 11, 2016; Published date: May 13, 2016

Citation: Reis PEO, Roever L, Reis IFO. Endovascular Management of Acute Pulmonary Embolus. Journal of Vascular & Endo Surgery. 2016, 1:2. doi: 10.21767/2573-4482.100011

Visit for more related articles at Journal of Vascular and Endovascular Therapy

Keywords

Embolims; Treatment; Endovascular

Introduction

The first pulmonary embolectomy was done by Friedrich Trendelenburg in 1908 with no survivor and the first successful pulmonary embolectomy was in 1924 [1-3]. Now approximately 630 000 pulmonary embolisms (PE) occur yearly in the United States, resulting in 300 000 deaths [4-8].

The symptoms most often found in patients with PTE are: dyspnea, chest pain, pleuritic chest pain, cough, swelling of the legs, pain in legs, hemoptysis, palpitations, wheezing, chest pain type angina, syncope, hemodynamic instability, acute right ventricular failure, cardiogenic shock and the main imaging methods used in the diagnosis are ventilation-perfusion scintigraphy, pulmonary angiography and computed tomography (CT) (Figure 1).

vascular-endovascular-surgery-red-arrows

Figure 1: Chest CT-The red arrows shows massive pulmonary embolism from both pulmonary arteries.

The guidelines for the management of TEP addresses the risk factors for major and minor, as the relative risk requiring the occurrence of TEP (Table 1). Deep vein thrombosis (DVT) in the lower limbs is mainly responsible for pulmonary thromboembolism (PTE) [9].

Major (relative risk between 5 and 20) Lower (relative risk between 2 and 4)
• Surgeries
Major abdominal surgery or pelvic/Hip prosthesis or knee/Post-operative ICU
• Obstetrical Events
End of pregnancy/Cesarean/Puerperium
• Lower members
Fractures/Varicose Veins
• Neoplasms
Abdominals/Pelvic/Metastatic
• Immobilization
Hospitalization/Nursing Homes
• Others
TVP confirmed prior
• Cardiovascular
Congenital heart disease/Heart failure
Congestive/Hypertension/vein thrombosis
Superficial/Central venous catheter
• Estrogen therapy
Contraceptive/Hormone replacement
• Others
COPD/Neurological Diseases/Hidden Neoplasms/Thrombotic disorders/Prolonged Travel/Obesity/Inflammatory disease intestinal/Nephrotic syndrome/Chronic Dialysis/Myeloproliferative disorders/Hemoglobinuria/Behcet's Disease

Table 1: Risk factors for pulmonary embolism.

With the advancement of new endovascular technologies we have available various catheters for thrombectomy and thrombolysis to treat patients with massive or submassive PE [10-12]. Tajima et al. [10] described a modified rotating pigtail catheter to treat critical pulmonary emboli. Pulmonary embolectomy for massive PE could have good results [13,14]. However with endovascular procedures PE can be treated quickly and efficiently without a thoracotomy [15-18].

CHEST Guideline and Expert Panel Report published in 2016 suggest the use of catheter-removing thrombus or mechanical interventions in patients with acute pulmonary embolism associated with hypotension, and have a high risk of bleeding or failed systemic thrombolysis, or shock presence that it can progress to death before systemic thrombolysis [19].

In conclusion we agree that probably some patients PE should be treated to remove the embolus and if the PE does not affect right ventricular function the patient will generally do well with anticoagulation.

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